{"id":3578,"date":"2026-02-12T16:09:24","date_gmt":"2026-02-12T13:09:24","guid":{"rendered":"https:\/\/wiki.dev.voka.io\/diseases\/uncategorized\/hiperparatireoidismo\/"},"modified":"2026-03-16T13:02:49","modified_gmt":"2026-03-16T10:02:49","slug":"hiperparatireoidismo","status":"publish","type":"diseases_post","link":"https:\/\/wiki.dev.voka.io\/pt\/doencas\/endocrinologia\/hiperparatireoidismo\/","title":{"rendered":"Hiperparatireoidismo: classifica\u00e7\u00e3o, patog\u00eanese, apresenta\u00e7\u00e3o cl\u00ednica, diagn\u00f3stico e tratamento"},"content":{"rendered":"<p>O hiperparatireoidismo \u00e9 um dist\u00farbio end\u00f3crino causado pela produ\u00e7\u00e3o excessiva de horm\u00f4nio da paratireoide (tamb\u00e9m chamado de paratorm\u00f4nio), levando a um dist\u00farbio no metabolismo do c\u00e1lcio e f\u00f3sforo e ao desenvolvimento de hipercalcemia, osteopenia, disfun\u00e7\u00e3o renal e comprometimento do sistema cardiovascular. \u00c9 a terceira doen\u00e7a end\u00f3crina mais comum (depois de diabetes mellitus e de doen\u00e7as da tireoide), observada com mais frequ\u00eancia em mulheres. <\/p>\n<p><a href=\"https:\/\/catalog.voka.io\/en\/models\/862dbbd9-7cc2-476f-9800-793443a3b8a7\/7c26be50-7df9-4002-aff7-76dc2ff5bdd0\/fa3dd8fc-9df4-4ad4-a3be-2f2e91df06f2\/94f7a892-704d-4e14-886f-647e10260b39\" target=\"_blank\" rel=\"noreferrer noopener nofollow\">Modelos 3D<\/a> de variantes de hiperparatireoidismo:<\/p>\n<div class=\"carousel-block\">\n<div class=\"swiper carousel-swiper carousel-swiper-block_69e0ffae8e11f\">\n<div class=\"swiper-wrapper\">\n<div class=\"swiper-slide\"><img decoding=\"async\" class=\"carousel-image\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/articles\/en\/endocrinology\/hyperparathyroidism\/1-primary-hyperparathyroidism.webp\" alt=\"Hiperparatireoidismo prim\u00e1rio\"><span class=\"small-text-article text-main-text-color\">Hiperparatireoidismo prim\u00e1rio<\/span><\/div>\n<div class=\"swiper-slide\"><img decoding=\"async\" class=\"carousel-image\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/articles\/en\/endocrinology\/hyperparathyroidism\/2-secondary-hyperparathyroidism.webp\" alt=\"Hiperparatireoidismo secund\u00e1rio\"><span class=\"small-text-article text-main-text-color\">Hiperparatireoidismo secund\u00e1rio<\/span><\/div>\n<div class=\"swiper-slide\"><img decoding=\"async\" class=\"carousel-image\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/articles\/en\/endocrinology\/hyperparathyroidism\/3-tertiary-hyperparathyroidism.webp\" alt=\"Hiperparatireoidismo terci\u00e1rio\"><span class=\"small-text-article text-main-text-color\">Hiperparatireoidismo terci\u00e1rio<\/span><\/div>\n<\/div>\n<\/div>\n<div class=\"swiper-article-carousel__controls\">\n<div class=\"swiper-article-carousel__pagination-container swiper-article-carousel__pagination-container-block_69e0ffae8e11f\"><\/div>\n<div class=\"swiper-article-carousel__arrows\"><button class=\"swiper__arrow swiper__arrow--prev carousel-swiper-button-prev swiper__arrow swiper__arrow--prev carousel-swiper-button-prev-block_69e0ffae8e11f\"><svg xmlns=\"http:\/\/www.w3.org\/2000\/svg\" width=\"40\" height=\"40\" viewbox=\"0 0 40 40\" fill=\"none\"><path d=\"M20 3.75C16.7861 3.75 13.6443 4.70305 10.972 6.48862C8.29969 8.27419 6.21689 10.8121 4.98696 13.7814C3.75704 16.7507 3.43524 20.018 4.06225 23.1702C4.68926 26.3224 6.23692 29.2179 8.50952 31.4905C10.7821 33.7631 13.6776 35.3107 16.8298 35.9378C19.982 36.5648 23.2493 36.243 26.2186 35.013C29.1879 33.7831 31.7258 31.7003 33.5114 29.028C35.297 26.3557 36.25 23.2139 36.25 20C36.2455 15.6916 34.5319 11.561 31.4855 8.51454C28.439 5.46806 24.3084 3.75455 20 3.75ZM23.3844 25.3656C23.5005 25.4818 23.5926 25.6196 23.6555 25.7714C23.7184 25.9231 23.7507 26.0858 23.7507 26.25C23.7507 26.4142 23.7184 26.5769 23.6555 26.7286C23.5926 26.8804 23.5005 27.0182 23.3844 27.1344C23.2682 27.2505 23.1304 27.3426 22.9786 27.4055C22.8269 27.4683 22.6643 27.5007 22.5 27.5007C22.3358 27.5007 22.1731 27.4683 22.0214 27.4055C21.8696 27.3426 21.7318 27.2505 21.6156 27.1344L15.3656 20.8844C15.2494 20.7683 15.1572 20.6304 15.0943 20.4787C15.0314 20.3269 14.999 20.1643 14.999 20C14.999 19.8357 15.0314 19.6731 15.0943 19.5213C15.1572 19.3696 15.2494 19.2317 15.3656 19.1156L21.6156 12.8656C21.7318 12.7495 21.8696 12.6574 22.0214 12.5945C22.1731 12.5317 22.3358 12.4993 22.5 12.4993C22.6643 12.4993 22.8269 12.5317 22.9786 12.5945C23.1304 12.6574 23.2682 12.7495 23.3844 12.8656C23.5005 12.9818 23.5926 13.1196 23.6555 13.2714C23.7184 13.4231 23.7507 13.5858 23.7507 13.75C23.7507 13.9142 23.7184 14.0769 23.6555 14.2286C23.5926 14.3804 23.5005 14.5182 23.3844 14.6344L18.0172 20L23.3844 25.3656Z\" fill=\"#B5C0CD\"><\/path><\/svg><\/button><button class=\"swiper__arrow swiper__arrow--next carousel-swiper-button-next swiper__arrow swiper__arrow--next carousel-swiper-button-next-block_69e0ffae8e11f\" tabindex=\"0\" aria-label=\"Next slide\"><svg xmlns=\"http:\/\/www.w3.org\/2000\/svg\" width=\"40\" height=\"40\" viewbox=\"0 0 40 40\" fill=\"none\"><path d=\"M20 3.75C16.7861 3.75 13.6443 4.70305 10.972 6.48862C8.29969 8.27419 6.21689 10.8121 4.98696 13.7814C3.75704 16.7507 3.43524 20.018 4.06225 23.1702C4.68926 26.3224 6.23692 29.2179 8.50952 31.4905C10.7821 33.7631 13.6776 35.3107 16.8298 35.9378C19.982 36.5648 23.2493 36.243 26.2186 35.013C29.1879 33.7831 31.7258 31.7003 33.5114 29.028C35.297 26.3557 36.25 23.2139 36.25 20C36.2455 15.6916 34.5319 11.561 31.4855 8.51454C28.439 5.46806 24.3084 3.75455 20 3.75ZM23.3844 25.3656C23.5005 25.4818 23.5926 25.6196 23.6555 25.7714C23.7184 25.9231 23.7507 26.0858 23.7507 26.25C23.7507 26.4142 23.7184 26.5769 23.6555 26.7286C23.5926 26.8804 23.5005 27.0182 23.3844 27.1344C23.2682 27.2505 23.1304 27.3426 22.9786 27.4055C22.8269 27.4683 22.6643 27.5007 22.5 27.5007C22.3358 27.5007 22.1731 27.4683 22.0214 27.4055C21.8696 27.3426 21.7318 27.2505 21.6156 27.1344L15.3656 20.8844C15.2494 20.7683 15.1572 20.6304 15.0943 20.4787C15.0314 20.3269 14.999 20.1643 14.999 20C14.999 19.8357 15.0314 19.6731 15.0943 19.5213C15.1572 19.3696 15.2494 19.2317 15.3656 19.1156L21.6156 12.8656C21.7318 12.7495 21.8696 12.6574 22.0214 12.5945C22.1731 12.5317 22.3358 12.4993 22.5 12.4993C22.6643 12.4993 22.8269 12.5317 22.9786 12.5945C23.1304 12.6574 23.2682 12.7495 23.3844 12.8656C23.5005 12.9818 23.5926 13.1196 23.6555 13.2714C23.7184 13.4231 23.7507 13.5858 23.7507 13.75C23.7507 13.9142 23.7184 14.0769 23.6555 14.2286C23.5926 14.3804 23.5005 14.5182 23.3844 14.6344L18.0172 20L23.3844 25.3656Z\" fill=\"#B5C0CD\"><\/path><\/svg><\/button><\/div>\n<\/div>\n<\/div>\n<p><script defer>\n    document.addEventListener('DOMContentLoaded', function() {\n        const imagesCarouselSwiper = new Swiper('.carousel-swiper-block_69e0ffae8e11f', {\n            slidesPerView: 1,\n            spaceBetween: 20,\n            loop: true,\n            pagination: {\n                el: '.swiper-article-carousel__pagination-container-block_69e0ffae8e11f',\n                clickable: true,\n            },\n            navigation: {\n                nextEl: '.carousel-swiper-button-next-block_69e0ffae8e11f',\n                prevEl: '.carousel-swiper-button-prev-block_69e0ffae8e11f',\n            },\n        })\n    })\n<\/script><\/p>\n<h2 class=\"wp-block-heading\" id=\"classificacao-do-hiperparatireoidismo\">Classifica\u00e7\u00e3o do hiperparatireoidismo<\/h2>\n<p>Dependendo da causa:<\/p>\n<ul class=\"wp-block-list\">\n<li>Prim\u00e1rio;<\/li>\n<li>Secund\u00e1rio;<\/li>\n<li>Terci\u00e1rio.<\/li>\n<\/ul>\n<p>Classifica\u00e7\u00e3o pelo curso cl\u00ednico:<\/p>\n<ul class=\"wp-block-list\">\n<li>Subcl\u00ednico ou assintom\u00e1tico;<\/li>\n<li>Manifesto;<\/li>\n<li>Hiperparatireoidismo agudo, ou crise de hiperparatireoide. <\/li>\n<\/ul>\n<h3 class=\"wp-block-heading\" id=\"hiperparatireoidismo-primario-etiologia-e-patogenese\">Hiperparatireoidismo prim\u00e1rio: etiologia e patog\u00eanese<\/h3>\n<figure class=\"wp-block-image size-large\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/articles\/en\/endocrinology\/hyperparathyroidism\/1-primary-hyperparathyroidism.webp\" alt=\"Hiperparatireoidismo prim\u00e1rio\n\"><figcaption class=\"wp-element-caption\">Hiperparatireoidismo prim\u00e1rio: <a href=\"https:\/\/catalog.voka.io\/en\/models\/862dbbd9-7cc2-476f-9800-793443a3b8a7\/0e08e7b2-6ea5-4221-8185-708345f19be1\/89143e3f-f3ef-4993-abfe-f48e417eb1f5\/1a51952f-ccd8-437e-aaa9-ac95e45ae66f\" target=\"_blank\" rel=\"noreferrer noopener nofollow\">modelo 3D<\/a><br \/><\/figcaption><\/figure>\n<p>O hiperparatireoidismo prim\u00e1rio \u00e9 uma condi\u00e7\u00e3o caracterizada pelo aumento da produ\u00e7\u00e3o de horm\u00f4nio da paratireoide.<\/p>\n<p>Na maioria esmagadora dos casos (80 a 85%), ele \u00e9 causado por um adenoma de uma das gl\u00e2ndulas paratireoides. Menos frequentemente, ocorre hiperplasia das gl\u00e2ndulas paratireoides e carcinoma das gl\u00e2ndulas paratireoides (menos de 1%). Desenvolve-se, na maioria das vezes, devido \u00e0 patologia de uma das gl\u00e2ndulas paratireoides. <\/p>\n<p>O adenoma da gl\u00e2ndula paratireoide \u00e9 caracterizado pelo desenvolvimento de autonomia funcional, levando ao aumento da produ\u00e7\u00e3o de horm\u00f4nio da paratireoide.<\/p>\n<p>O adenoma da gl\u00e2ndula paratireoide pode ser espor\u00e1dico ou geneticamente determinado (at\u00e9 10% dos casos).<\/p>\n<p>O adenoma da gl\u00e2ndula paratireoide pode ocorrer nas seguintes s\u00edndromes heredit\u00e1rias:<\/p>\n<ul class=\"wp-block-list\">\n<li>S\u00edndromes de Neoplasia End\u00f3crina M\u00faltipla (NEM) tipos 1, 2a e 4;<\/li>\n<li>S\u00edndrome de hiperparatireoidismo associado a tumor de mand\u00edbula ou maxila (HPT-JT);<\/li>\n<li>Hipercalcemia hipocalci\u00farica familiar;<\/li>\n<li>Hiperparatireoidismo isolado familiar.<\/li>\n<\/ul>\n<p>O horm\u00f4nio paratireoide desempenha um papel crucial no metabolismo do c\u00e1lcio-f\u00f3sforo atrav\u00e9s dos seguintes mecanismos:<\/p>\n<ol class=\"wp-block-list\">\n<li>Aumento da reabsor\u00e7\u00e3o do tecido \u00f3sseo, resultando na libera\u00e7\u00e3o de c\u00e1lcio e f\u00f3sforo para o soro sangu\u00edneo;<\/li>\n<li>Redu\u00e7\u00e3o da excre\u00e7\u00e3o de c\u00e1lcio na urina por meio do aumento da reabsor\u00e7\u00e3o renal;<\/li>\n<li>Ativa\u00e7\u00e3o da vitamina D 1-alfa-hidroxilase, convertendo 25-hidroxivitamina D na forma ativa (1,25-dihidroxivitamina D), o que aumenta a absor\u00e7\u00e3o de c\u00e1lcio e f\u00f3sforo no trato gastrointestinal.<\/li>\n<\/ol>\n<p>Al\u00e9m disso, o horm\u00f4nio da paratireoide inibe a reabsor\u00e7\u00e3o de f\u00f3sforo pelos t\u00fabulos renais, levando \u00e0 hiperfosfat\u00faria. Este mecanismo mant\u00e9m os n\u00edveis de f\u00f3sforo no soro dentro ou abaixo da faixa normal. <\/p>\n<p>Em pacientes com hiperparatireoidismo prim\u00e1rio, o efeito do aumento da reabsor\u00e7\u00e3o de c\u00e1lcio pode ser compensado por hipercalcemia elevada. Em tais casos, ser\u00e1 detectada hipercalci\u00faria.<\/p>\n<p>Assim, os pacientes com hiperparatireoidismo prim\u00e1rio s\u00e3o caracterizados por:<\/p>\n<ul class=\"wp-block-list\">\n<li>N\u00edveis aumentados do horm\u00f4nio da paratireoide;<\/li>\n<li>Hipercalcemia (devido \u00e0 reabsor\u00e7\u00e3o do tecido \u00f3sseo e aumento da absor\u00e7\u00e3o de c\u00e1lcio e f\u00f3sforo no trato gastrointestinal);<\/li>\n<li>Normo- ou hipofosfatemia;<\/li>\n<li>Hiperfosfat\u00faria;<\/li>\n<li>Hipercalci\u00faria com altos n\u00edveis de c\u00e1lcio no soro.<\/li>\n<\/ul>\n<h3 class=\"wp-block-heading\" id=\"hiperparatireoidismo-secundario-etiologia-e-patogenese\">Hiperparatireoidismo secund\u00e1rio: etiologia e patog\u00eanese<\/h3>\n<figure class=\"wp-block-image size-large\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/articles\/en\/endocrinology\/hyperparathyroidism\/2-secondary-hyperparathyroidism.webp\" alt=\"Hiperparatireoidismo secund\u00e1rio\n\"><figcaption class=\"wp-element-caption\">Hiperparatireoidismo secund\u00e1rio: <a href=\"https:\/\/catalog.voka.io\/en\/models\/862dbbd9-7cc2-476f-9800-793443a3b8a7\/0e08e7b2-6ea5-4221-8185-708345f19be1\/89143e3f-f3ef-4993-abfe-f48e417eb1f5\/1ffdabd5-0312-474d-b659-b06938feee88\" target=\"_blank\" rel=\"noreferrer noopener nofollow\">modelo 3D<\/a><\/figcaption><\/figure>\n<p>O hiperparatireoidismo secund\u00e1rio desenvolve-se no contexto da doen\u00e7a renal cr\u00f4nica (DRC) como uma compensa\u00e7\u00e3o pela hipocalcemia prolongada.<\/p>\n<p>Seu desenvolvimento envolve os seguintes mecanismos:<\/p>\n<ol class=\"wp-block-list\">\n<li>A disfun\u00e7\u00e3o renal leva a uma diminui\u00e7\u00e3o nos n\u00edveis de 1,25-dihidroxivitamina D (ativado a partir de 25-hidroxivitamina D nos t\u00fabulos renais proximais), resultando em menor absor\u00e7\u00e3o de c\u00e1lcio e f\u00f3sforo no trato gastrointestinal e no desenvolvimento de hipocalcemia;<\/li>\n<li>A hipocalcemia estimula receptores sens\u00edveis ao c\u00e1lcio das gl\u00e2ndulas paratireoides, levando ao aumento da produ\u00e7\u00e3o do horm\u00f4nio da paratireoide;<\/li>\n<li>O dist\u00farbio de excre\u00e7\u00e3o de f\u00f3sforo em doen\u00e7as renais cr\u00f4nicas leva \u00e0 hiperfosfatemia, ativando diretamente as gl\u00e2ndulas paratireoides e aumentando a produ\u00e7\u00e3o do horm\u00f4nio da paratireoide.<\/li>\n<\/ol>\n<p>Os mecanismos mencionados resultam na estimula\u00e7\u00e3o cont\u00ednua da fun\u00e7\u00e3o das gl\u00e2ndulas paratireoides e, como consequ\u00eancia, no desenvolvimento de hiperplasia.<\/p>\n<p>Assim, o hiperparatireoidismo secund\u00e1rio \u00e9 caracterizado por:<\/p>\n<ul class=\"wp-block-list\">\n<li>N\u00edveis aumentados do horm\u00f4nio da paratireoide;<\/li>\n<li>Hiperfosfatemia;<\/li>\n<li>Hipocalcemia, raramente normocalcemia.<\/li>\n<\/ul>\n<h3 class=\"wp-block-heading\" id=\"hiperparatireoidismo-terciario-etiologia-e-patogenese\">Hiperparatireoidismo terci\u00e1rio: etiologia e patog\u00eanese<\/h3>\n<figure class=\"wp-block-image size-large\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/articles\/en\/endocrinology\/hyperparathyroidism\/3-tertiary-hyperparathyroidism.webp\" alt=\"Hiperparatireoidismo terci\u00e1rio\"><figcaption class=\"wp-element-caption\">Hiperparatireoidismo terci\u00e1rio: <a href=\"https:\/\/catalog.voka.io\/en\/models\/862dbbd9-7cc2-476f-9800-793443a3b8a7\/0e08e7b2-6ea5-4221-8185-708345f19be1\/89143e3f-f3ef-4993-abfe-f48e417eb1f5\/f02c1141-2059-4632-a645-8e542b398502\" target=\"_blank\" rel=\"noreferrer noopener nofollow\">modelo 3D<\/a><\/figcaption><\/figure>\n<p>O hiperparatireoidismo terci\u00e1rio desenvolve-se em associa\u00e7\u00e3o com hiperparatireoidismo secund\u00e1rio de longa dura\u00e7\u00e3o, levando \u00e0 transforma\u00e7\u00e3o adenomatonosa das gl\u00e2ndulas paratireoides com o desenvolvimento de autonomia funcional.<\/p>\n<p>O hiperparatireoidismo terci\u00e1rio \u00e9 caracterizado por:<\/p>\n<ul class=\"wp-block-list\">\n<li>N\u00edveis significativamente aumentados do horm\u00f4nio da paratireoide;<\/li>\n<li>Hipercalcemia;<\/li>\n<li>Hiperfosfatemia;<\/li>\n<li>Hiperfosfaturia e hipercalci\u00faria.<\/li>\n<\/ul>\n<h2 class=\"wp-block-heading\" id=\"apresentacao-clinica\">Apresenta\u00e7\u00e3o cl\u00ednica <\/h2>\n<p>O hiperparatireoidismo pode ocorrer em tr\u00eas variantes:<\/p>\n<ul class=\"wp-block-list\">\n<li>Subcl\u00ednico ou assintom\u00e1tico;<\/li>\n<li>Manifesto; <\/li>\n<li>Agudo, ou crise hipercalcemica.<\/li>\n<\/ul>\n<p>Atualmente, na maioria dos casos, o hiperparatireoidismo \u00e9 detectado na forma subcl\u00ednica por meio de estudos cl\u00ednicos e laboratoriais de rotina, e os pacientes n\u00e3o relatam quaisquer queixas no momento do diagn\u00f3stico.<\/p>\n<h3 class=\"wp-block-heading\" id=\"hiperparatireoidismo-manifesto\">Hiperparatireoidismo manifesto<\/h3>\n<p>Os sintomas precoces do hiperparatireoidismo s\u00e3o inespec\u00edficos. Entre elas est\u00e3o:<\/p>\n<ul class=\"wp-block-list\">\n<li>Fadiga aumentada;<\/li>\n<li>Fraqueza muscular;<\/li>\n<li>Unhas quebradi\u00e7as, queda de cabelo;<\/li>\n<li>Dor nas articula\u00e7\u00f5es e m\u00fasculos, especialmente ap\u00f3s atividades f\u00edsicas;<\/li>\n<li>Labilidade emocional, preju\u00edzo de mem\u00f3ria.<\/li>\n<\/ul>\n<p>O hiperparatireoidismo de longa data leva ao desenvolvimento de osteoporose, cujos sintomas s\u00e3o:<\/p>\n<ul class=\"wp-block-list\">\n<li>Dor nos ossos, articula\u00e7\u00f5es e m\u00fasculos, que piora com o esfor\u00e7o f\u00edsico;<\/li>\n<li>Fragilidade \u00f3ssea, levando a fraturas mesmo com carga di\u00e1ria (tamb\u00e9m conhecidas como fraturas patol\u00f3gicas), que cicatrizam muito lentamente, frequentemente com forma\u00e7\u00e3o proeminente de calo \u00f3sseo, deformidade do osso fraturado e forma\u00e7\u00e3o de articula\u00e7\u00f5es falsas;<\/li>\n<li>Eros\u00e3o do esmalte dent\u00e1rio, afrouxamento e perda de dentes;<\/li>\n<li>Tens\u00e3o muscular e c\u00e3ibras, especialmente \u00e0 noite;<\/li>\n<li>Altera\u00e7\u00f5es posturais e redu\u00e7\u00e3o da altura devido a uma cifose tor\u00e1cica mais evidente.<\/li>\n<\/ul>\n<p>Al\u00e9m das manifesta\u00e7\u00f5es de osteoporose, o hiperparatireoidismo \u00e9 caracterizado pelos seguintes sintomas causados por hipercalcemia prolongada:<\/p>\n<ol class=\"wp-block-list\">\n<li>Dos rins e sistema urin\u00e1rio:<\/li>\n<\/ol>\n<ul class=\"wp-block-list\">\n<li>Nefrolit\u00edase;<\/li>\n<li>Poli\u00faria;<\/li>\n<li>Polidipsia.<\/li>\n<\/ul>\n<ol start=\"2\" class=\"wp-block-list\">\n<li>Sistema cardiovascular:<\/li>\n<\/ol>\n<ul class=\"wp-block-list\">\n<li>Calcifica\u00e7\u00e3o dos vasos sangu\u00edneos e v\u00e1lvulas card\u00edacas;<\/li>\n<li>Hipertens\u00e3o;<\/li>\n<li>Encurtamento do intervalo QT, dist\u00farbios de condu\u00e7\u00e3o levando a arritmias potencialmente fatais.<\/li>\n<\/ul>\n<ol start=\"3\" class=\"wp-block-list\">\n<li>Sistema gastrointestinal:<\/li>\n<\/ol>\n<ul class=\"wp-block-list\">\n<li>Perda de apetite;<\/li>\n<li>N\u00e1usea e v\u00f4mitos;<\/li>\n<li>Obstipa\u00e7\u00e3o;<\/li>\n<li>Dor abdominal e flatul\u00eancia;<\/li>\n<li>Desenvolvimento de \u00falceras p\u00e9pticas de v\u00e1rias localiza\u00e7\u00f5es, propensas a sangramentos e recorr\u00eancias.<\/li>\n<\/ul>\n<ol start=\"4\" class=\"wp-block-list\">\n<li>Sistema nervoso:<\/li>\n<\/ol>\n<ul class=\"wp-block-list\">\n<li>Labilidade emocional: ansiedade, irritabilidade, depress\u00e3o, apatia;<\/li>\n<li>Preju\u00edzo de mem\u00f3ria;<\/li>\n<li>Dist\u00farbios do sono;<\/li>\n<li>Dores de cabe\u00e7a.<\/li>\n<\/ul>\n<ol start=\"5\" class=\"wp-block-list\">\n<li>Calcifica\u00e7\u00e3o da conjuntiva (s\u00edndrome do olho vermelho);<\/li>\n<\/ol>\n<ol start=\"6\" class=\"wp-block-list\">\n<li>Forma\u00e7\u00e3o de oss\u00edculos em tecidos moles.<\/li>\n<\/ol>\n<h3 class=\"wp-block-heading\" id=\"hiperparatireoidismo-agudo-crise-hipercalcemica\">Hiperparatireoidismo agudo (crise hipercalc\u00eamica)<\/h3>\n<p>Uma crise hipercalc\u00eamica (ou crise hiperparatireoide) \u00e9 uma condi\u00e7\u00e3o que causa risco de vida e se desenvolve em pacientes com hiperparatireoidismo pr\u00e9-existente. Caracterizada por um r\u00e1pido aumento acentuado nos n\u00edveis de c\u00e1lcio s\u00e9rico acima de 3,5 mmol\/L, com os n\u00edveis de horm\u00f4nio da paratireoide geralmente excedendo a norma em mais de 20 vezes. <\/p>\n<p>Os fatores que podem provocar o desenvolvimento de crises incluem doen\u00e7as infecciosas, desidrata\u00e7\u00e3o e imobiliza\u00e7\u00e3o prolongada. A mortalidade varia de 60 a 90% e requer terapia imediata em unidades de terapia intensiva.<\/p>\n<p>Sintomas de crise hipercalc\u00eamica:<\/p>\n<ul class=\"wp-block-list\">\n<li>V\u00f4mito severo, desidrata\u00e7\u00e3o;<\/li>\n<li>Dor abdominal;<\/li>\n<li>Fraqueza muscular acentuada;<\/li>\n<li>Letargia, perda de consci\u00eancia, at\u00e9 o desenvolvimento de coma.<\/li>\n<\/ul>\n<h2 class=\"wp-block-heading\" id=\"diagnostico\">Diagn\u00f3stico <\/h2>\n<p>M\u00e9todos principais de exame:<\/p>\n<ol class=\"wp-block-list\">\n<li>Hist\u00f3rico e exame f\u00edsico;<\/li>\n<\/ol>\n<ol start=\"2\" class=\"wp-block-list\">\n<li>Os diagn\u00f3sticos laboratoriais s\u00e3o o m\u00e9todo principal para identificar o hiperparatireoidismo.<\/li>\n<\/ol>\n<p>Em uma bioqu\u00edmica sangu\u00ednea, devem-se determinar os n\u00edveis de horm\u00f4nio da paratireoide, c\u00e1lcio (total e ionizado), f\u00f3sforo, fosfatase alcalina (aumentada devido \u00e0 reabsor\u00e7\u00e3o \u00f3ssea) e vitamina D. Al\u00e9m disso, os n\u00edveis de excre\u00e7\u00e3o de c\u00e1lcio e f\u00f3sforo na urina s\u00e3o avaliados (teste de urina de 24 horas).<\/p>\n<p>Indicadores laboratoriais din\u00e2micos para v\u00e1rias variantes de hiperparatireoidismo:<\/p>\n<figure class=\"wp-block-table table-to-cards\">\n<table class=\"has-fixed-layout\">\n<thead>\n<tr>\n<th class=\"has-text-align-center\" data-align=\"center\">Indicadores s\u00e9ricos<\/th>\n<th class=\"has-text-align-center\" data-align=\"center\">Hiperparatireoidismo prim\u00e1rio<\/th>\n<th class=\"has-text-align-center\" data-align=\"center\">Hiperparatireoidismo secund\u00e1rio<\/th>\n<th class=\"has-text-align-center\" data-align=\"center\">Hiperparatireoidismo terci\u00e1rio<\/th>\n<\/tr>\n<\/thead>\n<tbody>\n<tr>\n<td class=\"has-text-align-center\" data-align=\"center\">Horm\u00f4nio da paratireoide<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Elevada<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Elevada<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Significativamente elevado<\/td>\n<\/tr>\n<tr>\n<td class=\"has-text-align-center\" data-align=\"center\">C\u00e1lcio<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Elevada<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Normal ou diminu\u00eddo<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Elevada<\/td>\n<\/tr>\n<tr>\n<td class=\"has-text-align-center\" data-align=\"center\">F\u00f3sforo<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Normal ou diminu\u00eddo<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Elevada<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Elevada<\/td>\n<\/tr>\n<tr>\n<td class=\"has-text-align-center\" data-align=\"center\">Fosfatase alcalina<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Elevada<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Elevada<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Elevada<\/td>\n<\/tr>\n<tr>\n<td class=\"has-text-align-center\" data-align=\"center\">Vitamina D<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Baixo<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Baixo<\/td>\n<td class=\"has-text-align-center\" data-align=\"center\">Baixo<\/td>\n<\/tr>\n<\/tbody>\n<\/table>\n<\/figure>\n<p>M\u00e9todos de imagem das paratireoides:<\/p>\n<ul class=\"wp-block-list\">\n<li>Ultrassom (m\u00e9todo de imagem principal);<\/li>\n<li>Cintilografia das paratireoides com Tc99m [tecn\u00e9cio-99m] (permite visualiza\u00e7\u00e3o da atividade funcional das paratireoides);<\/li>\n<li>M\u00e9todos de imagem adicionais podem incluir tomografia computadorizada e resson\u00e2ncia magn\u00e9tica do pesco\u00e7o e mediastino.<\/li>\n<\/ul>\n<p>M\u00e9todos de exame adicionais:<\/p>\n<ul class=\"wp-block-list\">\n<li>Densitometria para avaliar a densidade mineral \u00f3ssea;<\/li>\n<li>Ultrassom dos rins e trato urin\u00e1rio para identificar nefrolit\u00edase;<\/li>\n<li>Radiografia de ossos e articula\u00e7\u00f5es.<\/li>\n<\/ul>\n<p>Sinais radiol\u00f3gicos caracter\u00edsticos do hiperparatireoidismo:<\/p>\n<ul class=\"wp-block-list\">\n<li>Reabsor\u00e7\u00e3o \u00f3ssea subperiostal;<\/li>\n<li>Reabsor\u00e7\u00e3o subcondral;<\/li>\n<li>Reabsor\u00e7\u00e3o subenteseal;<\/li>\n<li>Reabsor\u00e7\u00e3o intracortical;<\/li>\n<li>Acrooste\u00f3lise;<\/li>\n<li>Osteopenia; <\/li>\n<li>Condrocalcinose;<\/li>\n<li>Sinal do sal e pimenta na radiografia do cr\u00e2nio;<\/li>\n<li>Calcifica\u00e7\u00e3o de tecido mole (no hiperparatireoidismo secund\u00e1rio e terci\u00e1rio);<\/li>\n<li>Osteosclerose.<\/li>\n<\/ul>\n<div class=\"social-banner-block\">\n<div class=\"social-banner-content\">\n<p class=\"h5-title text-black\">Encontra mais conte\u00fados cientificamente exactos nas nossas redes sociais<\/p>\n<p><span class=\"social-banner-text text-grey\">Subscreve e n\u00e3o percas os recursos mais recentes<\/span><\/p>\n<div class=\"social-links-wrapper\"><a class=\"social-icon-link\" href=\"https:\/\/www.facebook.com\/VOKA3DAnatomyAndPathology\/\" target=\"_blank\" rel=\"nofollow noopener\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/common\/Social%20Icons\/facebook.svg\" alt=\"social link\"><\/a><a class=\"social-icon-link\" href=\"https:\/\/www.instagram.com\/voka.io\/\" target=\"_blank\" rel=\"nofollow noopener\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/common\/Social%20Icons\/insta.svg\" alt=\"social link\"><\/a><a class=\"social-icon-link\" href=\"https:\/\/www.linkedin.com\/company\/voka-io\/posts\/?feedView=all\" target=\"_blank\" rel=\"nofollow noopener\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/common\/Social%20Icons\/linkedin.svg\" alt=\"social link\"><\/a><a class=\"social-icon-link\" href=\"https:\/\/www.youtube.com\/@vokaio\" target=\"_blank\" rel=\"nofollow noopener\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/common\/Social%20Icons\/youtube.svg\" alt=\"social link\"><\/a><a class=\"social-icon-link\" href=\"https:\/\/www.pinterest.com\/voka3danatomyandpathology\/\" target=\"_blank\" rel=\"nofollow noopener\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/common\/Social%20Icons\/pinterest.svg\" alt=\"social link\"><\/a><a class=\"social-icon-link\" href=\"https:\/\/www.tiktok.com\/@voka.io\" target=\"_blank\" rel=\"nofollow noopener\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/common\/Social%20Icons\/tiktok.svg\" alt=\"social link\"><\/a><a class=\"social-icon-link\" href=\"https:\/\/discord.gg\/7ejUpq8DRR\" target=\"_blank\" rel=\"nofollow noopener\"><img decoding=\"async\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/common\/Social%20Icons\/discord.svg\" alt=\"social link\"><\/a><\/div>\n<\/div>\n<p><img decoding=\"async\" class=\"social-banner-image\" loading=\"lazy\" src=\"https:\/\/storage.googleapis.com\/dev_wiki_voka_io_303011\/common\/social-media-banner-mobile-image.webp\" alt=\"Banner background\"><\/div>\n<h2 class=\"wp-block-heading\" id=\"tratamento-do-hiperparatireoidismo\">Tratamento do hiperparatireoidismo<\/h2>\n<h3 class=\"wp-block-heading\" id=\"hiperparatireoidismo-primario\">Hiperparatireoidismo prim\u00e1rio<\/h3>\n<p>O principal m\u00e9todo de tratamento \u00e9 cir\u00fargico. Isso permite a normaliza\u00e7\u00e3o dos n\u00edveis de paratorm\u00f4nio, c\u00e1lcio e f\u00f3sforo no menor tempo poss\u00edvel, reduzindo assim o risco de complica\u00e7\u00f5es. <\/p>\n<p>A opera\u00e7\u00e3o envolve a remo\u00e7\u00e3o da gl\u00e2ndula paratireoide adenomatosa (parotidectomia). Este \u00e9 o padr\u00e3o ouro para a terapia do hiperparatireoidismo prim\u00e1rio.<\/p>\n<p>Ap\u00f3s a opera\u00e7\u00e3o, \u00e9 poss\u00edvel o desenvolvimento da s\u00edndrome da \u201cfome \u00f3ssea\u201d, que \u00e9 uma diminui\u00e7\u00e3o persistente (mais de 4 dias) dos n\u00edveis de c\u00e1lcio no soro sangu\u00edneo. Esta s\u00edndrome \u00e9 causada por uma diminui\u00e7\u00e3o acentuada dos n\u00edveis de paratorm\u00f4nio ap\u00f3s a cirurgia, levando ao aumento da atividade osteobl\u00e1stica e captura de c\u00e1lcio e f\u00f3sforo para a forma\u00e7\u00e3o do tecido \u00f3sseo. <\/p>\n<p>Em um exame de bioqu\u00edmica do sangue, s\u00e3o detectados hipocalcemia, hipofosfatemia, hipomagnesemia e n\u00edveis elevados de fosfatase alcalina. Esta s\u00edndrome \u00e9 mais comum em cirurgias para hiperparatireoidismo secund\u00e1rio. <\/p>\n<p>Pacientes com contraindica\u00e7\u00f5es ao tratamento cir\u00fargico podem receber terapia conservadora em casos de hipercalcemia leve e aus\u00eancia de complica\u00e7\u00f5es. Os seguintes medicamentos s\u00e3o usados para o tratamento do hiperparatireoidismo:<\/p>\n<ul class=\"wp-block-list\">\n<li><strong>Vitamina D.<\/strong> A dosagem \u00e9 selecionada individualmente para atingir o n\u00edvel recomendado de 25-hidroxivitamina D no soro do sangue (30 ng\/ml), contribuindo para a redu\u00e7\u00e3o da reabsor\u00e7\u00e3o do tecido \u00f3sseo.<\/li>\n<li><strong>Bifosfonatos (\u00e1cido clodr\u00f4nico, \u00e1cido pamidr\u00f4nico, \u00e1cido ibandr\u00f4nico, \u00e1cido zoledr\u00f4nico)<\/strong>. Inibe a reabsor\u00e7\u00e3o do tecido \u00f3sseo.<\/li>\n<li><strong>Calcimim\u00e9ticos (cinacalcete, etelcalcitide)<\/strong>. Reduz significativamente os n\u00edveis de paratorm\u00f4nio e c\u00e1lcio no soro sangu\u00edneo, mas n\u00e3o afeta a reabsor\u00e7\u00e3o do tecido \u00f3sseo.<\/li>\n<li><strong>Denosumabe (inibidor do ligante do RANK)<\/strong>. Inibe a reabsor\u00e7\u00e3o do tecido \u00f3sseo. Estes medicamentos podem ser prescritos tanto isoladamente quanto em combina\u00e7\u00e3o para controle adequado dos n\u00edveis de paratorm\u00f4nio, c\u00e1lcio e densidade mineral \u00f3ssea.<\/li>\n<li><strong>Ingest\u00e3o oral de fosfato<\/strong>. Diminui os n\u00edveis de c\u00e1lcio no soro do sangue.<\/li>\n<\/ul>\n<p>Al\u00e9m disso, os pacientes que recebem terapia conservadora n\u00e3o s\u00e3o aconselhados a restringir a ingest\u00e3o diet\u00e9tica de c\u00e1lcio, pois a defici\u00eancia de c\u00e1lcio pode levar \u00e0 estimula\u00e7\u00e3o das gl\u00e2ndulas paratiroides e um aumento nos n\u00edveis de paratorm\u00f4nio.<\/p>\n<h3 class=\"wp-block-heading\" id=\"hiperparatireoidismo-secundario-e-terciario\">Hiperparatireoidismo secund\u00e1rio e terci\u00e1rio<\/h3>\n<p>O principal m\u00e9todo de tratamento \u00e9 conservador. A terapia medicamentosa visa manter e, se poss\u00edvel, normalizar os n\u00edveis de paratorm\u00f4nio, c\u00e1lcio e f\u00f3sforo. <\/p>\n<p>Al\u00e9m dos medicamentos usados no hiperparatireoidismo prim\u00e1rio, tamb\u00e9m s\u00e3o usados ativadores de receptores de vitamina D no tratamento do hiperparatireoidismo secund\u00e1rio e terci\u00e1rio: <\/p>\n<ol class=\"wp-block-list\">\n<li>Metab\u00f3litos da vitamina D (colecalciferol, ergocalciferol);<\/li>\n<li>An\u00e1logos da vitamina D (calcitriol, paricalcitol, alfecalcidol, doxercalciferol).<\/li>\n<\/ol>\n<p>Se a terapia m\u00e9dica for ineficaz, o tratamento cir\u00fargico \u00e9 indicado. A extens\u00e3o da interven\u00e7\u00e3o cir\u00fargica \u00e9 determinada individualmente, com base nos n\u00edveis de paratorm\u00f4nio, c\u00e1lcio, f\u00f3sforo, no estado som\u00e1tico geral do paciente e na presen\u00e7a de complica\u00e7\u00f5es do hiperparatireoidismo. <\/p>\n<p>As seguintes opera\u00e7\u00f5es podem ser realizadas:<\/p>\n<ul class=\"wp-block-list\">\n<li><strong>Paratiroidectomia subtotal (deixando uma parte do tecido da gl\u00e2ndula paratiroide)<\/strong>. Risco m\u00ednimo de hipocalcemia permanente, maior risco de recorr\u00eancia.<\/li>\n<li><strong>Paratireoidectomia total sem autotransplante<\/strong>. Risco m\u00ednimo de recorr\u00eancia; o m\u00e9todo tem limita\u00e7\u00f5es devido ao desenvolvimento de hipocalcemia persistente no per\u00edodo p\u00f3s-operat\u00f3rio, que muitas vezes \u00e9 mal controlada com medicamentos.<\/li>\n<li><strong>Paratireoidectomia total com autotransplante de tecido da gl\u00e2ndula paratiroide nos m\u00fasculos do antebra\u00e7o<\/strong>. O risco de recorr\u00eancia \u00e9 menor do que com paratireoidectomia subtotal, com um risco moderado de hipocalcemia persistente no per\u00edodo p\u00f3s-operat\u00f3rio. <\/li>\n<\/ul>\n<h3 class=\"wp-block-heading\" id=\"crise-de-hiperparatireoidismo\">Crise de hiperparatireoidismo<\/h3>\n<p>O tratamento \u00e9 realizado em uma unidade de terapia intensiva e ressuscita\u00e7\u00e3o e inclui o seguinte:<\/p>\n<ul class=\"wp-block-list\">\n<li>Terapia de infus\u00e3o com grandes volumes de solu\u00e7\u00e3o salina para reabastecer o volume sangu\u00edneo circulante (reidrata\u00e7\u00e3o);<\/li>\n<li>Furosemida para for\u00e7ar a diurese (excre\u00e7\u00e3o de c\u00e1lcio);<\/li>\n<li>A di\u00e1lise \u00e9 poss\u00edvel, especialmente em pacientes com insufici\u00eancia renal cr\u00f4nica;<\/li>\n<li>Administra\u00e7\u00e3o de bifosfonatos, calcimim\u00e9ticos, denosumabe, calcitonina.<\/li>\n<\/ul>\n<p>Ap\u00f3s a estabiliza\u00e7\u00e3o da condi\u00e7\u00e3o, o tratamento cir\u00fargico \u00e9 indicado (paratireoidectomia para hiperparatireoidismo prim\u00e1rio e paratireoidectomia subtotal ou total para hiperparatireoidismo secund\u00e1rio ou terci\u00e1rio).<\/p>\n<div>\n<h2 class=\"faq-title h2-article\" id=\"faq\">FAQ<\/h2>\n<div class=\"faq-section\">\n<div class=\"faq-item faq-answer-hidden\">\n<div class=\"question-block\">\n<div class=\"faq-question\">\n<p class=\" text-black h5-title\">1. O que \u00e9 hiperparatireoidismo e quais s\u00e3o suas consequ\u00eancias? <\/p>\n<\/div>\n<div class=\"faq-answer text-main-text-color main-text-medium\">O hiperparatireoidismo \u00e9 um dist\u00farbio end\u00f3crino associado \u00e0 secre\u00e7\u00e3o excessiva de paratorm\u00f4nio (PTH). Isso leva \u00e0 libera\u00e7\u00e3o de c\u00e1lcio do tecido \u00f3sseo (reabsor\u00e7\u00e3o), causando osteoporose e risco de fraturas. Um excesso de c\u00e1lcio no sangue (hipercalcemia) contribui para o desenvolvimento de nefrolit\u00edase (c\u00e1lculos renais) e calcifica\u00e7\u00e3o vascular e card\u00edaca.<\/div>\n<\/div>\n<div class=\"expand-button-wrapper\"><button class=\"text-accent expand-button\">+<\/button><\/div>\n<\/div>\n<div class=\"faq-item faq-answer-hidden\">\n<div class=\"question-block\">\n<div class=\"faq-question\">\n<p class=\" text-black h5-title\">2. Quais s\u00e3o os principais sintomas cl\u00ednicos do hiperparatireoidismo em mulheres? <\/p>\n<\/div>\n<div class=\"faq-answer text-main-text-color main-text-medium\">A doen\u00e7a \u00e9 diagnosticada tr\u00eas vezes mais frequentemente em mulheres. O quadro cl\u00ednico inclui fraqueza geral severa, dor nos ossos e articula\u00e7\u00f5es, bem como osteoporose. Os sintomas frequentemente apresentam-se como n\u00e3o espec\u00edficos, imitando depress\u00e3o, comprometimento da mem\u00f3ria ou s\u00edndrome da fadiga cr\u00f4nica.<\/div>\n<\/div>\n<div class=\"expand-button-wrapper\"><button class=\"text-accent expand-button\">+<\/button><\/div>\n<\/div>\n<div class=\"faq-item faq-answer-hidden\">\n<div class=\"question-block\">\n<div class=\"faq-question\">\n<p class=\" text-black h5-title\">3. Quais exames laboratoriais s\u00e3o necess\u00e1rios para verificar o diagn\u00f3stico? <\/p>\n<\/div>\n<div class=\"faq-answer text-main-text-color main-text-medium\">Diagn\u00f3sticos laboratoriais abrangentes s\u00e3o conduzidos para confirmar o diagn\u00f3stico. Indicadores s\u00e9ricos chave: n\u00edvel de horm\u00f4nio da paratireoide (paratorm\u00f4nio, ou PTH), c\u00e1lcio total e ionizado, f\u00f3sforo, fosfatase alcalina e vitamina D. Tamb\u00e9m \u00e9 necess\u00e1ria uma an\u00e1lise di\u00e1ria de urina para excre\u00e7\u00e3o de c\u00e1lcio e f\u00f3sforo para avaliar o metabolismo mineral.<\/div>\n<\/div>\n<div class=\"expand-button-wrapper\"><button class=\"text-accent expand-button\">+<\/button><\/div>\n<\/div>\n<div class=\"faq-item faq-answer-hidden\">\n<div class=\"question-block\">\n<div class=\"faq-question\">\n<p class=\" text-black h5-title\">4. \u00c9 poss\u00edvel uma terapia conservadora eficaz para hiperparatireoidismo prim\u00e1rio? <\/p>\n<\/div>\n<div class=\"faq-answer text-main-text-color main-text-medium\">No hiperparatireoidismo prim\u00e1rio causado por adenoma, o padr\u00e3o ouro e \u00fanico m\u00e9todo de cura continua sendo a remo\u00e7\u00e3o cir\u00fargica da gl\u00e2ndula. A terapia conservadora (bifosfonatos, calcimim\u00e9ticos) \u00e9 usada como medida paliativa quando a cirurgia \u00e9 contraindicada ou como parte da prepara\u00e7\u00e3o para a interven\u00e7\u00e3o cir\u00fargica.<\/div>\n<\/div>\n<div class=\"expand-button-wrapper\"><button class=\"text-accent expand-button\">+<\/button><\/div>\n<\/div>\n<div class=\"faq-item faq-answer-hidden\">\n<div class=\"question-block\">\n<div class=\"faq-question\">\n<p class=\" text-black h5-title\">5. Por que o hiperparatireoidismo secund\u00e1rio se desenvolve no contexto de doen\u00e7a renal? <\/p>\n<\/div>\n<div class=\"faq-answer text-main-text-color main-text-medium\">Na doen\u00e7a renal cr\u00f4nica (DRC), a s\u00edntese de vitamina D ativa e a excre\u00e7\u00e3o de f\u00f3sforo s\u00e3o prejudicadas, levando a uma diminui\u00e7\u00e3o nos n\u00edveis de c\u00e1lcio no sangue. Isso desencadeia um mecanismo compensat\u00f3rio: as gl\u00e2ndulas paratiroides come\u00e7am a trabalhar em modo intensificado, o que causa sua hiperplasia e eleva\u00e7\u00e3o persistente dos n\u00edveis de paratorm\u00f4nio ao longo do tempo.<\/div>\n<\/div>\n<div class=\"expand-button-wrapper\"><button class=\"text-accent expand-button\">+<\/button><\/div>\n<\/div>\n<div class=\"faq-item faq-answer-hidden\">\n<div class=\"question-block\">\n<div class=\"faq-question\">\n<p class=\" text-black h5-title\">6. Quais princ\u00edpios de terapia diet\u00e9tica devem ser seguidos no hiperparatireoidismo? <\/p>\n<\/div>\n<div class=\"faq-answer text-main-text-color main-text-medium\">N\u00e3o \u00e9 aconselhado aos pacientes a restri\u00e7\u00e3o rigorosa de c\u00e1lcio em sua dieta, pois sua defici\u00eancia pode estimular ainda mais a secre\u00e7\u00e3o de paratorm\u00f4nio. O monitoramento dos n\u00edveis de vitamina D \u00e9 importante. No entanto, no hiperparatireoidismo secund\u00e1rio causado por patologia renal, \u00e9 de import\u00e2ncia cr\u00edtica limitar alimentos ricos em f\u00f3sforo.<\/div>\n<\/div>\n<div class=\"expand-button-wrapper\"><button class=\"text-accent expand-button\">+<\/button><\/div>\n<\/div>\n<\/div>\n<p><script type=\"application\/ld+json\">\n    {\n    \"@context\": \"https:\/\/schema.org\",\n    \"@type\": \"FAQPage\",\n    \"mainEntity\": [\n        {\n            \"@type\": \"Question\",\n            \"name\": \"O que \u00e9 hiperparatireoidismo e quais s\u00e3o suas consequ\u00eancias? \",\n            \"acceptedAnswer\": {\n                \"@type\": \"Answer\",\n                \"text\": \"O hiperparatireoidismo \u00e9 um dist\u00farbio end\u00f3crino associado \u00e0 secre\u00e7\u00e3o excessiva de paratorm\u00f4nio (PTH). Isso leva \u00e0 libera\u00e7\u00e3o de c\u00e1lcio do tecido \u00f3sseo (reabsor\u00e7\u00e3o), causando osteoporose e risco de fraturas. Um excesso de c\u00e1lcio no sangue (hipercalcemia) contribui para o desenvolvimento de nefrolit\u00edase (c\u00e1lculos renais) e calcifica\u00e7\u00e3o vascular e card\u00edaca.\"\n            }\n        },\n        {\n            \"@type\": \"Question\",\n            \"name\": \"Quais s\u00e3o os principais sintomas cl\u00ednicos do hiperparatireoidismo em mulheres? \",\n            \"acceptedAnswer\": {\n                \"@type\": \"Answer\",\n                \"text\": \"A doen\u00e7a \u00e9 diagnosticada tr\u00eas vezes mais frequentemente em mulheres. O quadro cl\u00ednico inclui fraqueza geral severa, dor nos ossos e articula\u00e7\u00f5es, bem como osteoporose. Os sintomas frequentemente apresentam-se como n\u00e3o espec\u00edficos, imitando depress\u00e3o, comprometimento da mem\u00f3ria ou s\u00edndrome da fadiga cr\u00f4nica.\"\n            }\n        },\n        {\n            \"@type\": \"Question\",\n            \"name\": \"Quais exames laboratoriais s\u00e3o necess\u00e1rios para verificar o diagn\u00f3stico? \",\n            \"acceptedAnswer\": {\n                \"@type\": \"Answer\",\n                \"text\": \"Diagn\u00f3sticos laboratoriais abrangentes s\u00e3o conduzidos para confirmar o diagn\u00f3stico. Indicadores s\u00e9ricos chave: n\u00edvel de horm\u00f4nio da paratireoide (paratorm\u00f4nio, ou PTH), c\u00e1lcio total e ionizado, f\u00f3sforo, fosfatase alcalina e vitamina D. Tamb\u00e9m \u00e9 necess\u00e1ria uma an\u00e1lise di\u00e1ria de urina para excre\u00e7\u00e3o de c\u00e1lcio e f\u00f3sforo para avaliar o metabolismo mineral.\"\n            }\n        },\n        {\n            \"@type\": \"Question\",\n            \"name\": \"\u00c9 poss\u00edvel uma terapia conservadora eficaz para hiperparatireoidismo prim\u00e1rio? \",\n            \"acceptedAnswer\": {\n                \"@type\": \"Answer\",\n                \"text\": \"No hiperparatireoidismo prim\u00e1rio causado por adenoma, o padr\u00e3o ouro e \u00fanico m\u00e9todo de cura continua sendo a remo\u00e7\u00e3o cir\u00fargica da gl\u00e2ndula. A terapia conservadora (bifosfonatos, calcimim\u00e9ticos) \u00e9 usada como medida paliativa quando a cirurgia \u00e9 contraindicada ou como parte da prepara\u00e7\u00e3o para a interven\u00e7\u00e3o cir\u00fargica.\"\n            }\n        },\n        {\n            \"@type\": \"Question\",\n            \"name\": \"Por que o hiperparatireoidismo secund\u00e1rio se desenvolve no contexto de doen\u00e7a renal? \",\n            \"acceptedAnswer\": {\n                \"@type\": \"Answer\",\n                \"text\": \"Na doen\u00e7a renal cr\u00f4nica (DRC), a s\u00edntese de vitamina D ativa e a excre\u00e7\u00e3o de f\u00f3sforo s\u00e3o prejudicadas, levando a uma diminui\u00e7\u00e3o nos n\u00edveis de c\u00e1lcio no sangue. Isso desencadeia um mecanismo compensat\u00f3rio: as gl\u00e2ndulas paratiroides come\u00e7am a trabalhar em modo intensificado, o que causa sua hiperplasia e eleva\u00e7\u00e3o persistente dos n\u00edveis de paratorm\u00f4nio ao longo do tempo.\"\n            }\n        },\n        {\n            \"@type\": \"Question\",\n            \"name\": \"Quais princ\u00edpios de terapia diet\u00e9tica devem ser seguidos no hiperparatireoidismo? \",\n            \"acceptedAnswer\": {\n                \"@type\": \"Answer\",\n                \"text\": \"N\u00e3o \u00e9 aconselhado aos pacientes a restri\u00e7\u00e3o rigorosa de c\u00e1lcio em sua dieta, pois sua defici\u00eancia pode estimular ainda mais a secre\u00e7\u00e3o de paratorm\u00f4nio. O monitoramento dos n\u00edveis de vitamina D \u00e9 importante. No entanto, no hiperparatireoidismo secund\u00e1rio causado por patologia renal, \u00e9 de import\u00e2ncia cr\u00edtica limitar alimentos ricos em f\u00f3sforo.\"\n            }\n        }\n    ]\n}<\/script><\/div>\n<div class=\"sources-list-block sources-list-hidden\" id=\"referencias\">\n<div class=\"sources-list-content\">\n<div class=\"sources-list-title\">\n<p class=\"small-text-bold text-black sources-list-title-text\">Refer\u00eancias <\/p>\n<div class=\"sources-expand-button-wrapper-mobile\">\n<div class=\"sources-expand-button\"><svg width=\"32\" height=\"32\" viewbox=\"0 0 32 32\" fill=\"none\" xmlns=\"http:\/\/www.w3.org\/2000\/svg\"><path d=\"M8 12L16 20L24 12\" stroke=\"#8C9AAB\" stroke-width=\"2\" stroke-linecap=\"round\" stroke-linejoin=\"round\"><\/path><\/svg><\/div>\n<\/div>\n<\/div>\n<div class=\"sources-list-items\">\n<div class=\"source-item\">\n<p class=\"main-text-semibold text-black\">1.<\/p>\n<div class=\"source-item-content\">\n<p class=\"main-text-semibold text-black\"><cite>VOKA 3D Anatomy & Pathology \u2013 Complete Anatomy and Pathology 3D Atlas (Atlas 3D completo de anatomia e patologia) [Internet]. VOKA 3D Anatomy & Pathology<\/cite><\/p>\n<p><span class=\"small-text-medium text-grey\">Dispon\u00edvel em: https:\/\/catalog.voka.io\/ <\/span><\/div>\n<\/div>\n<div class=\"source-item\">\n<p class=\"main-text-semibold text-black\">2.<\/p>\n<div class=\"source-item-content\">\n<p class=\"main-text-semibold text-black\"><cite>Helbing A, Leslie SW, Levine SN. Primary Hyperparathyroidism [Hiperparatireoidismo prim\u00e1rio]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing. <\/cite><\/p>\n<p><span class=\"small-text-medium text-grey\">Dispon\u00edvel em: https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK441895\/<\/span><\/div>\n<\/div>\n<div class=\"source-item\">\n<p class=\"main-text-semibold text-black\">3.<\/p>\n<div class=\"source-item-content\">\n<p class=\"main-text-semibold text-black\"><cite>Muppidi V, Meegada SR, Rehman A. Secondary Hyperparathyroidism [Hiperparatireoidismo secund\u00e1rio]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing. <\/cite><\/p>\n<p><span class=\"small-text-medium text-grey\">Dispon\u00edvel em: https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK557822\/ <\/span><\/div>\n<\/div>\n<div class=\"source-item\">\n<p class=\"main-text-semibold text-black\">4.<\/p>\n<div class=\"source-item-content\">\n<p class=\"main-text-semibold text-black\"><cite>Palumbo VD, Damiano G, Messina M, Fazzotta S, Lo Monte G, Lo Monte AI. Tertiary Hyperparathyroidism: a review [Hiperparatireoidismo terci\u00e1rio: uma revis\u00e3o]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing.<\/cite><\/p>\n<p><span class=\"small-text-medium text-grey\">Dispon\u00edvel em: https:\/\/pubmed.ncbi.nlm.nih.gov\/33956045\/ <\/span><\/div>\n<\/div>\n<div class=\"source-item\">\n<p class=\"main-text-semibold text-black\">5.<\/p>\n<div class=\"source-item-content\">\n<p class=\"main-text-semibold text-black\"><cite>Campos A. Hyperparathyroidism [Hiperparatireoidismo]. PubMed [Internet]. <\/cite><\/p>\n<p><span class=\"small-text-medium text-grey\">Dispon\u00edvel em: https:\/\/radiopaedia.org\/articles\/hyperparathyroidism<\/span><\/div>\n<\/div>\n<\/div>\n<\/div>\n<div class=\"sources-expand-button-wrapper\">\n<div class=\"sources-expand-button\"><svg width=\"32\" height=\"32\" viewbox=\"0 0 32 32\" fill=\"none\" xmlns=\"http:\/\/www.w3.org\/2000\/svg\"><path d=\"M8 12L16 20L24 12\" stroke=\"#8C9AAB\" stroke-width=\"2\" stroke-linecap=\"round\" stroke-linejoin=\"round\"><\/path><\/svg><\/div>\n<\/div>\n<\/div>\n","protected":false},"excerpt":{"rendered":"<p>O hiperparatireoidismo \u00e9 um dist\u00farbio end\u00f3crino causado pela produ\u00e7\u00e3o excessiva de horm\u00f4nio da paratireoide (tamb\u00e9m chamado de paratorm\u00f4nio), levando a um dist\u00farbio no metabolismo do c\u00e1lcio e f\u00f3sforo e ao desenvolvimento de hipercalcemia, osteopenia, disfun\u00e7\u00e3o renal e comprometimento do sistema cardiovascular. \u00c9 a terceira doen\u00e7a end\u00f3crina mais comum (depois de diabetes mellitus e de doen\u00e7as [&hellip;]<\/p>\n","protected":false},"author":9,"featured_media":0,"template":"","diseases_category":[321],"class_list":["post-3578","diseases_post","type-diseases_post","status-publish","hentry","diseases_category-endocrinologia"],"acf":[],"yoast_head":"<!-- This site is optimized with the Yoast SEO Premium plugin v25.0 (Yoast SEO v26.5) - https:\/\/yoast.com\/wordpress\/plugins\/seo\/ -->\n<title>Hiperparatireoidismo: classifica\u00e7\u00e3o, sintomas, diagn\u00f3stico e tratamento<\/title>\n<meta name=\"description\" content=\"Hiperparatireoidismo como dist\u00farbio end\u00f3crino: altera\u00e7\u00f5es no metabolismo do c\u00e1lcio e f\u00f3sforo. 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